Iodine and your thyroid - Stop The Thyroid Madness. Home » Iodine and your thyroid. Interest in iodine supplementation has grown in leaps and bounds over the years among thyroid patients!? What about the small amount of iodine in natural desiccated thyroid? Apparently not, say many iodine experts. It’s far less than is needed. Plus with table salt, the iodine evaporates, i. These results can be important in revealing whether you might need to supplement or not. Some have chosen to do an iodine patch test on the skin, but results vary so widely that the loading test is the preferred and the most accurate determination. Doctor’s Data, Inc. Hakalalabs. Flechas and you call to order one. Lugols comes in 2% and 5%. One drop of the 5% equals 6. Iodine deficiency is a major health issue. Learn more about the symptoms of iodine deficiency, so you can protect yourself and your family. Comparing 4 Different Methods Of Iodine Testing. There are numerous ways to test for an iodine deficiency. If the person begins with an iodine patch test. Are you iodine deficient? The iodine patch test offers a quick and easy way to tell. It's simple and anyone can do it, learn how right here! The iodine skin patch test consists of applying iodine solution to a small area of the arm, leg, or abdomen. The faster the yellow color of iodine disappears from the skin, the more iodine deficient the person tested. Iodine Deficiency Patch Test For AllergiesIodoral is measured into 1. These can be ordered off the net by searching for either. Janie uses Lugols in her morning drink. There are others. What about Nascent iodine? There are patient reports of being fine on it. But Lugol’s and Iodoral have many decades of studies of their efficacy, whereas there’s very little on Nascent. That alone is why certain iodine experts are not keen on Nascent. Additionally, Nascent. Many start these one month before introducing iodine. Because of the MTHFR mutation, some people can have high levels, and being on selenium came make that worse with side effects to match, such as hair loss and more. Thus, if one’s levels are high, the selenium is not being broken down for use, and it will be important to adequately treat the mutation.? Can it be bad for Hashimoto’s disease? Yes, there are some patients who have reported an aggravation of their Hashimoto’s symptoms after using iodine. This underscores the need for supporting nutrients as outlined above before starting on iodine! To read interesting details about the association between the aggravation of Hashimoto’s and the rise of antibodies, go here. So we leave this up to you, but encourage you to do a lot of research to learn more. What if I’m allergic to iodine? There are suggestions by iodine experts that what is seen as an “allergy to iodine” may actually be a reaction to the injected contrast dyes which serve to sharpen the pictures in medical imaging studies (i. With reactions to certain seafoods like shellfish that are also high in iodine, they state it’s more likely due to a reaction distinctive allergens, aka certain proteins, found in these seafoods. A new epidemic of iodine deficiency has occurred. If you use colored iodine, put a small patch. There are fairly simple ways to administer an iodine deficiency test. Reply to Matt P; Quote Matt P. Detoxified Iodine Skin Patch Test, Episode #405 Matt Monarch. Subscribe Subscribed Unsubscribe 58,302 58K. Iodine Skin Patch Test for Iodine Deficiency and Slow Thyroid - Duration: 9:00. One of many studies on this issue here. Steven Rothrock, MD states: . In a survey of patients presenting to a pediatric clinic with suspected seafood allergy, 9. In a study from 2. IV contrast (e. g. CT scans and heart catherizations) in those with shellfish allergy. The authors of this study called this practice antiquated and recommended that this myth . But other adrenal patients report that the proper use of the same supporting nutrients mentioned above overcame the problem, and they were even able to lower their HC. An. There is already proof that iodine supplementation removes fibrocystic breast disease (it definitely removed Janie Bowthorpe’s), and that can be carried over to the proposed idea that iodine supplementation might prevent breast cancer. See the book Breast Cancer and Iodine by Dr. David Derry. Iodine and thyroid: Many hypothyroid patients were hopeful that iodine use would. For most, though, this has not been the case. And if started early enough in one’s thyroid disease, yes, some have actually been able to avoid thyroid medications! Iodine and. My own husband has completely removed two pre- cancer growths on each side of his nose with one drop of iodine per side at bedtime and for several weeks. Some practitioners recommend quite high doses of iodine. Are there other opinions about those high doses? Yes, here’s one. So it’s up to you. Where can I talk to other patients about iodine use? It can pay to explore the use of iodine and decide for yourself. There are at least two good iodine groups mentioned. Give references to where you found your information.)Stephanie Buist’s free article on iodine. Go here for Part One, then after you read it, go. Iit also has 1/5 the RDA of iodine (1. Other brands here.)FACTS ABOUT IODINE AND AUTOIMMUNE THYROIDITIS by Guy E. Hashimoto published in the German language and in a German medical journal (1), his histological findings in four thyroid glands removed at surgery: numerous lymphoid follicles; extensive connective tissue formation; diffuse round cell infiltration; and significant changes of the acinar epithelium. He called this pathology of the thyroid “struma lymphomatosa”, but it became popular under the name “Hashimoto Thyroiditis”. At the time of Hashimoto’s publication, autoimmune thyroiditis was not observed in the U. S. Both conditions are chronic, progressing over time to hypothyroidism in a significant percentage of Patients (2). In several communities worldwide, an increased incidence of AIT was reported following implementation of. In areas of the United States where this relationship has been studied, mainly in the Great Lakes Region, a similar trend was reported. In 1. 96. 6 and 1. Weaver et al (4,5) from Ann Arbor Michigan reported: “The salient histopathological feature of the thyroid glands, removed at operation in a five- year period before iodine prophylaxis (1. It should be emphasized that the thyroid glands prior to the use of iodized salt were devoid of lymphocytes, and nodular colloid goiters with dense lymphocytic infiltrates were found after the introduction of iodized salt in 1. Furszyfer et al (6), from the Mayo Clinic, studied the average annual incidence of Hashimoto’s thyroiditis among women of Olmsted County, Minnesota during 3 consecutive periods covering 3. They found the incidence to be higher in women 4. However, in both groups, there was a progressive increase in the incidence of Hashimoto’s thyroiditis over time. During the 3 periods evaluated, that is 1. Hashimoto’s per 1. For women 4. 0 years and older, the average annual incidence over the same 3 periods were: 1. It is important to point out that the Mayo Clinic study started 1. Therefore,even during the first decade of observation, the prevalence of autoimmune thyroiditis was already significant. Again, it must be emphasized that prior to the implementation of iodized salt as observed by Weaver, et al,(4. US, even though the Lugol solution and potassium iodide were used extensively in medical practice at that time in daily amount two orders of magnitude greater than the average intake of iodide from table salt. It is of interest to note that prior to iodization of salt, AIT was almost non- existent in the USA, although Lugol solution and potassium iodide were used extensively in medical practice in amounts 2 orders of magnitude greater than the average daily amount ingested from iodized salt (2). This suggests that inadequate iodide intake aggravated by goitrogens, not excess iodide, was the cause of this condition. Experimentally induced autoimmune thyroiditis in laboratory animals by acutely administered iodide required the use of antithyroid drugs, essentially goitrogens, to produce these effects (7- 1. These goitrogens induced thyroid hyperplasia and iodide deficiency. Bagchi et al (1. 1) and Many et al (1. We have previously proposed a mechanism for the oxidative damage caused by low levels of iodide combined with antithyroid drugs (2): Inadequate iodide supply to the thyroid gland, aggravated by goitrogens, activates the thyroid peroxydase (TPO) system through elevated TSH, low levels of iodinated lipids, and high cytosolic free calcium, resulting in excess production of H2. O2. The excess H2. O2 production is evidenced by the fact that antioxidants used in Bagchi’s experiments did not interfere with the oxidation and organification of iodide and therefore neutralized only the excess oxydant (1. This H2. O2 production is above normal due to a deficient feedback system caused by high cytosolic calcium due to magnesium deficiency and low levels of iodinated lipids which requires for their synthesis iodide levels 2 orders of magnitude greater than the RDA for iodine (2). Once the low iodide supply is depleted, TPO in the presence of H2. O2 Molar and organic substrate reverts to its peroxydase function which is the primary function of haloperoxydases, causing oxidative damage to molecules nearest to the site of action: TPO and the substrate thyroglobulin (Tg). Oxydized TPO and Tg elicit an autoimmune reaction with production of antibodies against these altered proteins with subsequent damage to the apical membrane of the thyroid cells, resulting in the lymphocytic infiltration and in the clinical manifestations of Hashimoto’s thyroiditis. Eventually, the oxidative damage to the TPO results in deficient H2. O2 production. Hypothyroidism occurs in AIT when oxidation and organification of iodide in the thyroid gland become deficient enough to affect synthesis of thyroid hormones. In vitro studies with purified fractions of calf thyroid glands by De Groot et al (1. Molar confers protection to TPO against oxidative damage. To achieve peripheral levels of 1. Molar iodide, a human adult needs a daily amount of 5. De. Groot’s findings can be summarized as follows: 1. TPO is inactivated by H2. O2. 2. KI at 1. 0- 5 Molar protects TPO from oxidative damage. Potassium Bromide and Potassium Fluoride do not share this protective. Based on the above facts, it is obvious that iodine deficiency, not excess, is the cause of AIT. References 1) Hashimoto, H., Zur Kenntniss der lymphomatosen Veranderung der Schilddruse (Struma lymphomatosa). Chir., 9. 7: 2. 19- 2. Abraham, G. E., The safe and effective implementation of orthoiodosupplementation in medical practice. The Original Internist, 1. Gaitan, E., Nelson, N. C., Poole, G. V., Endemic Goiter and Endemic Thyroid Disorders. Surg., 1. 5: 2. 05- 2. Surg., 9. 8: 1. 83- 1. Surg., 9. 2: 7. 96- 8. Proc., 4. 5: 5. 86- 5. In Werner & Ingbar’s The Thyroid – Braverman LE and Utiger RD Editors, Lippincott Williams & Wilkins, 7. Lab Invest., 1. 3: 1.
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